Diabetes mellitus (DM) is increasingly recognized as an independent risk factor for falls and balance disorders in elderly populations. While diabetic neuropathy and retinopathy are well-established complications, the role of vestibular system dysfunction has been underappreciated....
✦ The floor
Discussion
Signed responses from readers of the wire.
Audiologists and vestibular specialists should be aware that patients with diabetes may have higher BPPV recurrence risk due to vestibular system damage; however, this review is preliminary and does not yet support changes to BPPV repositioning protocols or follow-up intervals.
Understanding the diabetes-vestibular axis could help clinicians identify high-risk patients for BPPV recurrence and eventually guide more targeted rehabilitation strategies for a growing diabetic elderly population.
- 01Diabetes mellitus damages the vestibular system through multiple mechanisms including microangiopathy and neuropathy.
- 02These mechanisms may contribute to otolith dysfunction, raising BPPV risk and recurrence in diabetic patients.
- 03Elderly patients with diabetes represent a particularly vulnerable population for recurrent BPPV.
- 04The review synthesizes existing literature but does not present new clinical trial data.
- 05Findings suggest a need for prospective studies examining BPPV recurrence rates specifically in diabetic cohorts.
Diabetes mellitus damages the vestibular system and increases risk of BPPV pathogenesis and recurrence.
studypartially supportedElderly populations with diabetes are at elevated risk for BPPV recurrence.
studypartially supported- PMID
- 42871655
- DOI
- 10.47176/mjiri.40.75.
- Journal
- Medical Journal of the Islamic Republic of Iran
- Publication type
- review
- Evidence level
- 5
- Population
- Literature review; no direct patient population studied; focus on elderly adults with diabetes mellitus
- Intervention
- Narrative synthesis of mechanisms linking diabetes mellitus to vestibular system damage
Primary outcomes
Mechanisms of vestibular system damage in diabetes mellitus; Implications for BPPV pathogenesis and recurrence risk