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Presbycusis Across the Lifespan: Genetic, Molecular, and Multi-Omics Contributions

A dispatch from PubMed — filed

Presbycusis, or age-related hearing loss (ARHL), is a multifactorial disorder characterized by a gradual, bilateral sensorineural decline in hearing sensitivity, predominantly affecting high-frequency sounds. It is one of the most common chronic conditions in the aging population and represents a major public health concern due to its high prevalence and progressive nature....

Clinical Takeaway

This is a foundational science review; no actionable change to current clinical practice is indicated, but it informs understanding of why hearing loss progresses with age and may guide future therapeutic targets.

Why It Matters

A multi-omics perspective on presbycusis could accelerate the identification of drug targets and genetic risk factors, laying groundwork for future preventive or therapeutic interventions for age-related hearing loss.

Key Points
  1. 01Presbycusis (age-related hearing loss) is a multifactorial condition driven by genetic, epigenetic, and molecular factors.
  2. 02Multi-omics approaches (genomics, transcriptomics, proteomics, metabolomics) are being applied to understand inner-ear ageing.
  3. 03The review synthesises current knowledge on bilateral sensorineural hearing decline across the lifespan.
  4. 04Identifying molecular pathways could point to future drug or gene therapy targets.
  5. 05No single genetic cause explains presbycusis; many interacting variants contribute.
Claims & Evidence

Presbycusis results from multifactorial genetic and molecular mechanisms rather than a single cause.

studysupported

Multi-omics data can reveal novel molecular contributors to age-related sensorineural hearing decline.

studypartially supported
Research metadata
PMID
42345621
DOI
10.3390/audiolres16030081.
Journal
Audiology Research
Publication type
review
Evidence level
5
Population
Literature review; no direct patient population
Intervention
Genetic, molecular, and multi-omics analysis of presbycusis mechanisms

Primary outcomes

Identification of genetic and molecular contributors to presbycusis; Synthesis of multi-omics findings relevant to age-related hearing loss

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