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From weeks to hours: rapid whole-genome sequencing reduces diagnostic odyssey in Menke-Hennekam syndrome-a case report

A dispatch from PubMed — filed

Menke-Hennekam syndrome (MKHK) is a rare autosomal dominant disorder caused by mutations in the CREBBP and EP300 genes. The absence of established diagnostic criteria and non-specific clinical manifestations complicate timely diagnosis and management....

Clinical Takeaway

No actionable change for routine audiology practice; this single case report illustrates the diagnostic potential of rapid whole-genome sequencing for rare syndromes with hearing involvement, but does not establish a new clinical standard.

Why It Matters

Rapid whole-genome sequencing is beginning to compress diagnostic timelines for rare syndromic conditions that include hearing loss, which could eventually reshape referral pathways in genetic audiology.

Key Points
  1. 01Rapid whole-genome sequencing cut diagnosis time from weeks to hours for Menke-Hennekam syndrome.
  2. 02Menke-Hennekam syndrome is caused by mutations in the CREBBP or EP300 genes and follows an autosomal dominant inheritance pattern.
  3. 03Published as a case report in Journal of Medical Case Reports (2026).
  4. 04Single-case evidence; findings cannot be generalised without further study.
  5. 05Highlights a growing role for genomic medicine in the workup of rare syndromic hearing loss.
Claims & Evidence

Rapid whole-genome sequencing reduced the diagnostic timeline for Menke-Hennekam syndrome from weeks to hours.

studypartially supported

Menke-Hennekam syndrome is a rare autosomal dominant disorder caused by CREBBP/EP300 mutations.

guidelinesupported
Research metadata
PMID
42251442
DOI
10.1186/s13256-026-06163-z.
Journal
Journal of Medical Case Reports
Publication type
case_report
Evidence level
4
Sample size
1
Population
Individual patient diagnosed with Menke-Hennekam syndrome
Intervention
Rapid whole-genome sequencing for syndromic diagnosis

Primary outcomes

Time to definitive genetic diagnosis; Identification of causative CREBBP/EP300 variant

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